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Specific pattern of inflammatory responses linked to airways obstruction in asthma
By Mark Cowen
01 July 2010
Mucosal Immunol 2010; 3: 399–409

MedWire News: Chronic airways obstruction is associated with decreased activation of T-helper (Th)1-like/cytotoxic and interferon pathways in children who have suffered an asthma attack.

Writing in the journal Mucosal Immunology, A Bosco (University of Arizona, Tucson, USA) and colleagues explain that many children with asthma have chronic airflow obstruction, as indicated by deficits in the FEV1/forced vital capacity (FVC) ratio, while others do not.

To investigate whether a specific pattern of inflammatory responses in the airways during acute asthma exacerbations is associated with chronic airway obstruction, the team studied sputum samples collected during an acute asthma attack from 40 children with mild-to-moderate persistent asthma.

Ten of these samples were selected from children with significant pre-exacerbation chronic airway obstruction (mean FEV1/FVC ratio=78.8%) and 10 from children without pre-exacerbation chronic airway obstruction (mean FEV1/FVC ratio=91.3%).

Gene co-expression network analysis of the samples revealed decreased activation of Th1-like/cytotoxic and interferon signaling pathways, involving 115 and 78 genes, respectively, in asthmatic children with chronic airway obstruction relative to those without chronic airway obstruction.

These associations were independent of atopy, the presence of picornavirus infections, and the use of medications at the onset of the exacerbation, the researchers note.

Bosco and team conclude: "Our findings for the first time provide a global perspective of the immunological networks that are operating during naturally occurring acute asthma exacerbations in children, and broadly characterize variations in these networks that are associated with deficits in baseline FEV1/FVC ratios."

However, they caution: "Sputum induction was not performed during severe exacerbations because it was deemed unsafe and unethical, and thus the mechanisms operating in severe exacerbations may be different from those reported herein."

MedWire (www.medwire-news.md) is an independent clinical news service provided by Current Medicine Group, a trading division of Springer Healthcare Limited. © Springer Healthcare Ltd; 2010

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