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High levels TGF-β1 increase risk for incident Type 2 diabetes
By Helen Albert
20 October 2009
Diabetes Care 2009; 32: 1921–1923

MedWire News: Individuals with high levels of the cytokine transforming growth factor (TGF)-β1 have an increased risk for Type 2 diabetes, show study results.

“Subclinical inflammation represents one important mechanism in the development of insulin resistance and β-cell dysfunction, and a systemic proinflammatory state is associated with increased risk for Type 2 diabetes,” say Wolfgang Koenig (University of Ulm Medical Center, Germany) and team.

“However, data on anti-inflammatory immune mediators are scarce,” they add.

Koenig and colleagues therefore tested the concentration of TGF-β1, a mainly immunosuppressive, anti-inflammatory cytokine, in 460 people with Type 2 diabetes and 1474 individuals without the condition who acted as controls.

The participants were enrolled in the population-based MONICA (MONItoring of Trends and Determinants in CArdiovascular Disease)/KORA (Cooperative Health Research in the Region of Augsburg) study.

As reported in the journal Diabetes Care, the researchers found that the mean level of TGF-β1 in Type 2 diabetes patients versus controls was 35.8 versus 35.2 ng/ml, a nonsignificant difference.

However, when participants were stratified into TGF-β1 tertiles 1, 2, and 3 with median values of 28.1, 35.0, and 42.7 ng/ml, respectively, those in tertile 3 had a 41% increased likelihood of having Type 2 diabetes compared with those in tertile 1.

Further adjustment for body mass index and other lifestyle factors did not substantially affect the results.

The results of this study corroborate a previous study’s finding that high levels of another anti-inflammatory cytokine interleukin-1 receptor antagonist (IL-1Ra) preceded the onset of Type 2 diabetes, say the authors.

“This new finding underlines that the immune activation during the development of Type 2 diabetes is complex and includes both pro- and anti-inflammatory mediators,” conclude Koenig et al.

“One explanation could be that elevated concentrations of TGF-β1 and IL-1Ra represent a counter regulation of the proinflammatory state that increases the risk for Type 2 diabetes,” they suggest.

MedWire (www.medwire-news.md) is an independent clinical news service provided by Current Medicine Group, a trading division of Springer Healthcare Limited. © Springer Healthcare Ltd; 2009

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